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GLP-1 receptor agonists work by: Stimulating insulin secretion from pancreatic beta cells in a glucose-dependent manner, which helps regulate blood sugar levels Slowing gastric emptying , meaning food remains in the stomach longer, promoting feelings of fullness Acting on appetite centres in the brain , particularly the hypothalamus, to reduce hunger signals and food cravings Decreasing glucagon secretion , which helps prevent excessive glucose production by the liver Most GLP-1 agonists are administered via subcutaneous injection, typically once weekly or once daily depending on the specific formulation, though oral semaglutide (Rybelsus®) is also available

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Overall, a single administration of in vivo genome editing-mediated scExe4 knock-in provides stable, long-term metabolic benefits, effectively restoring blood glucose homeostasis and preventing relapse of pre-diabetic symptoms in non-genetic DIO mice